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Amenorrhea
Amenorrhea

... Formation of the ♂ external genitalia requiers 5α REDUCTASE testosterone     dihydrotestosterone Formation of the internal wollfiane structures respond directly to testosterone External genitalia ♀ with mild musculinization Absent uterus At puberty   testosterone secretion  virilization ...
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Ch 17 Powerpoint
Ch 17 Powerpoint

... II in the lung, which causes aldosterone secretion which ↑blood volume & ↑in blood pressure. »A second target for angiotensin II is arteriole walls – they constrict which further increases blood pressure. ...
Chapter 45.
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Gynaecology – Dr. `Abeer – Lecture 1 – Physiology of Menstruation

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Endocrine Emergencies - Department of Library Services

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CHAPTER 1 3
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Chapter 16 Cholinesterase Inhibitors

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endocrine lectures

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MCQ-endocrine File
MCQ-endocrine File

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Endocrinology of reproduction I (Lecture 6 and 7 combined)
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The Endocrine System • Endocrine and nervous systems work

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Endocrinology of reproduction I (Lecture 6 and 7
Endocrinology of reproduction I (Lecture 6 and 7

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Endocrine Virtual Lab! AP Biology
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Associations between Sex Hormones, Thyroid

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The Endocrine and Reproductive Systems Question No. 1 of 10
The Endocrine and Reproductive Systems Question No. 1 of 10

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GENder Education and Care Interdisciplinary Support (GENECIS

... medical as well as mental health wellbeing during HRT. If these requirements are not met, HRT may be discontinued in the best interest and safety of your child. Before beginning HRT your child needs to undergo a thorough psychological and social evaluation performed by our GENECIS team. We also requ ...
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Chapter 17

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The Endocrine System
The Endocrine System

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Hyperandrogenism

Hyperandrogenism, or androgen excess, is a medical condition characterized by excessive levels of androgens in the body and the associated effects of these excessive levels of androgens.Hyperandrogenism is one of the primary symptoms of polycystic ovary syndrome (PCOS). In such cases, it presents with symptoms such as acne and seborrhea, is frequent in adolescent girls and is often associated with irregular menstrual cycles. In most instances, these symptoms are transient and reflect only the immaturity of the hypothalamic-pituitary-ovarian axis during the first years following menarche. Approximately three-quarters of patients with PCOS (by the diagnostic criteria of NIH/NICHD 1990) have evidence of hyperandrogenism, with free testosterone being the single most predictive marker with ~60% of patients demonstrating supranormal levels.Hyperandrogenism can also be the result of excessive production of adrenal or gonadal androgens by adrenal adenomas, carcinomas, or hyperplasia, Leydig cell tumors in men, and arrhenoblastomas in women.In women, signs and symptoms of hyperandrogenism frequently include acne, scalp hair loss (androgenic alopecia), excessive facial and body hair (hirsutism), atypically high libido, breast atrophy, and others. Collectively, these symptoms are described as virilization.Management of hyperandrogenism symptoms like androgenic alopecia, include the use of antiandrogens such as cyproterone acetate, spironolactone, and flutamide.
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