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TWELVE-LEAD INTERPRETATION By Leslie Hernandez, BS, NREMT-P, LP THE HEART • One heart • Two sides/Two arteries • Three layers • Four chambers/valves BLOOD FLOW THROUGH THE HEART – Blood Flow • From the Body – Right Atrium • To the Lungs – Right Ventricle • From the Lungs – Left Atrium • To the Body – Left Ventricle THE ELECTRICAL CONDUCTION SYSTEM • • • • • • • • SA Node Intranodal Pathways AV Junction AV Fibers Bundle of His Septum Bundle Branches Purkinje System The Electrical Conduction System THE ECG LEADS • ECG Leads – Bipolar • Leads I, II, and II – Unipolar • Leads aVR, aVL, and aVF – Precordial • V1, V2, V3, V4, V5, V6 LEAD PLACEMENT THE ELECTROCARDIOGRAM WAVES, INTERVAL, SEGMENTS AND COMPLEXES INTERVALS AND SEGMENTS – Time Intervals • P–R Interval (PRI) or P–Q Interval (PQI) 120–200 ms • QRS Interval 80–100 ms • S–T Segment isoelectric • Q–T Interval 360–440 ms P WAVE P-Waves and Atrial Enlargement THE PR INTERVAL 120-200 ms THE QRS COMPLEX THE T WAVE How to Measure ST-Segment Deviation THE THREE I’S OF THE ACS • S–T Segment Changes – Associated with Myocardial Infarctions • Ischemia • Injury • Infarct Two Rules to Follow Changes >1 mm (0.1 mV) are significant If They occur in two or more contiguous leads Patterns and Localization • Inferior – II, III, and aVF • Septal – V1 and V2 • Anterior – V3 and V4 • Lateral – I, aVL, V5, and V6 12-Lead ECG Variations in AMI and Angina Baseline Ischemia—tall or inverted T wave (infarct), ST segment may be depressed (angina) Injury—elevated ST segment, T wave may invert Infarction (Acute)—abnormal Q wave, ST segment may be elevated and T wave may be inverted Infarction (Age Unknown)—abnormal Q wave, ST segment and T wave returned to normal Conditions that Mimic Injury • Pericarditis – Diffuse or “global” ST-segment changes • Left bundle branch blocks – If possibly new, must treat as MI • Left ventricular hypertrophy – Causes ST-segment elevation • Early repolarization – Causes ST-segment elevation • Ventricular paced rhythms – Causes ST-segment elevation THE NORMAL TWELVE-LEAD ANTERIOR MI ANTEROLATERAL MI Inf. Wall Inj. Or Infarct with RCA II, III, aVF, and V4R: • Hypotension • Supranodal and AV nodal blocks • Atrial fibrillation / flutter • PAC’s • Significant NTG and MS hemodynamic hypersensitivity INFERIOR MI HEXAXIAL SYSTEM NORMAL AND ABNORMAL AXES -90o Indeterminate Axis Abnormal Left Axis (Pathologic) -30o 180o 0 Right Axis Normal Left Axis (Physiologic) o Normal QRS Axis +90o Axis Deviation Axis Normal Left EXTREME RIGHT Lead I QRS Positive Positive Negative Lead aVF QRS Positive Negative Negative Right Negative Positive AXIS DEVIATION • Left Axis Deviation – Abnormal finding. – Often associated with hypertension, valvular heart disease, and other disease processes. Left Axis Deviation - Causes • Left ventricular enlargement – Hypertension – Aortic stenosis – Ischemic heart disease • Left bundle branch block and left anterior fascicular block RIGHT AXIS DEVIATION • Right Axis Deviation – Abnormal finding. – Often associated with COPD and pulmonary hypertension. Right Axis Deviation - Causes • Right ventricular enlargement – – – – COPD Pulmonary embolism Congenital heart diseases Other diseases causing pulmonary hypertension and cor pulmonale • Right bundle branch block and left posterior fascicular block BUNDLE BRANCH BLOCKS • Conduction Abnormalities – Bundle Branch Blocks • Right Bundle Branch Block BUNDLE BRANCH BLOCKS • Conduction Abnormalities – The Turn-Signal Rule • QRS > 120 ms throughout the ECG. • Look at the QRS in V1. • Identify the J point. • Draw a horizontal line. • Triangle pointing up indicates RBBB. • Triangle pointing down indicates LBBB. LEFT BUNDLE BRANCH BLOCK RIGHT BUNDLE BRANCH BLOCK CONDUCTION ABNORMALITIES • Conduction Abnormalities – Hemiblocks • Left Anterior Hemiblock CONDUCTION ABNORMALITIES • Conduction Abnormalities – Hemiblocks • Left Posterior Hemiblock CHAMBER ENLARGEMENT Chamber Enlargement Atrial Enlargement Ventricular Hypertrophy Causes Right-sided enlargement and hypertrophy, usually secondary to long-term pulmonary disease. Left-sided enlargement and hypertrophy, usually secondary to long-term hypertension. RIGHT ATRIAL ENLARGMENT LEFT ATRIAL ENLARGEMENT RIGHT VENTRICULAR ENLARGEMENT LEFT VENTRICULAR ENLARGEMENT Time to Practice . . . .