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Blood Banking Theory and Component Therapy
Blood Banking Theory and Component Therapy

... Author(s): Robertson Davenport, M.D., 2009 License: Unless otherwise noted, this material is made available under the terms of the Creative Commons Attribution–Noncommercial–Share Alike 3.0 License: http://creativecommons.org/licenses/by-nc-sa/3.0/ We have reviewed this material in accordance with U ...
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Cardiovascular Physiology

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... (CLC) to honor an individual for outstanding service to United Blood Services (UBS). It is named for Bill Shover for his significant contribution as a leader to UBS and the community blood program. The award is presented annually in February at the United Blood Services’ Valentines for Life luncheon ...
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... • Individuals with type A blood will not produce antibodies against A-type antigens. • Individuals with type B blood will not produce antibodies against B-type antigens but will produce antibodies against A markers. • Type AB individuals will not produce antibodies against A or B markers, so can tol ...
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CIR 15-HV-013 Additional Blood Donations Form

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... cell, attaches to the hemoglobin, and is reduced to the trivalent (plus 3) state. This state is maintained until the red blood cell is sequestered by the spleen, at which time the chromium is released to the plasma and is readily excreted in the urine. In the trivalent state, chromium Cr51 is not re ...
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Hemorheology

Hemorheology, also spelled haemorheology (from the Greek ‘αἷμα, haima ""blood"" and rheology), or blood rheology, is the study of flow properties of blood and its elements of plasma and cells. Proper tissue perfusion can occur only when blood's rheological properties are within certain levels. Alterations of these properties play significant roles in disease processes. Blood viscosity is determined by plasma viscosity, hematocrit (volume fraction of red blood cell, which constitute 99.9% of the cellular elements) and mechanical properties of red blood cells. Red blood cells have unique mechanical behavior, which can be discussed under the terms erythrocyte deformability and erythrocyte aggregation. Because of that, blood behaves as a non-Newtonian fluid. As such, the viscosity of blood varies with shear rate. Blood becomes less viscous at high shear rates like those experienced in peak-systole. Contrarily, during end-diastole, blood moves more slowly and becomes thicker and stickier. Therefore, blood is a shear-thinning fluid.
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