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NADF CUSHING`S PAMPHLET - National Adrenal Diseases
NADF CUSHING`S PAMPHLET - National Adrenal Diseases

... When the source of excess cortisol production is a tumor of the adrenal gland itself, then it is not dependent on ACTH. The tumor makes cortisol on its own, and the other adrenal gland shrinks because ACTH production is suppressed. Adrenal cortex tumors can be benign (an adenoma), or malignant (a ca ...
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... optimal results. • Hormones: Much like cortisol, the production of most sex hormones follows a diurnal rhythm. This must be taken into account. The maximum hormonal output for most male and female hormones is present after awakening from sleep. When assessing a patient’s peak hormone values, collec ...
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Lecture 046 - Animal Hormones

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Endocrinology: Endocrine System Function Nervous vs. Endocrine
Endocrinology: Endocrine System Function Nervous vs. Endocrine

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Endocrine functions of the pituitary and pineal glands 1/20
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... • These individuals may BECOME obese because their hypothalamus interprets starvation and causes hyperphagia, when they just cant use glucose that’s there! • These individuals may BECOME very skinny if excess eating cannot keep up with fatty acid oxidation. Responses are extremely viable. • Hyperins ...
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Hyperandrogenism

Hyperandrogenism, or androgen excess, is a medical condition characterized by excessive levels of androgens in the body and the associated effects of these excessive levels of androgens.Hyperandrogenism is one of the primary symptoms of polycystic ovary syndrome (PCOS). In such cases, it presents with symptoms such as acne and seborrhea, is frequent in adolescent girls and is often associated with irregular menstrual cycles. In most instances, these symptoms are transient and reflect only the immaturity of the hypothalamic-pituitary-ovarian axis during the first years following menarche. Approximately three-quarters of patients with PCOS (by the diagnostic criteria of NIH/NICHD 1990) have evidence of hyperandrogenism, with free testosterone being the single most predictive marker with ~60% of patients demonstrating supranormal levels.Hyperandrogenism can also be the result of excessive production of adrenal or gonadal androgens by adrenal adenomas, carcinomas, or hyperplasia, Leydig cell tumors in men, and arrhenoblastomas in women.In women, signs and symptoms of hyperandrogenism frequently include acne, scalp hair loss (androgenic alopecia), excessive facial and body hair (hirsutism), atypically high libido, breast atrophy, and others. Collectively, these symptoms are described as virilization.Management of hyperandrogenism symptoms like androgenic alopecia, include the use of antiandrogens such as cyproterone acetate, spironolactone, and flutamide.
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